question archive I am trying to find information regarding this scenario From course NURS 6501 Advanced pathophysiology: In the week's case study, a 27 year old was presented with a history of substance abuse and was found unresponsive by EMS after being called by the patient's roommate
I am trying to find information regarding this scenario From course NURS 6501 Advanced pathophysiology: In the week's case study, a 27 year old was presented with a history of substance abuse and was found unresponsive by EMS after being called by the patient's roommate
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I am trying to find information regarding this scenario From course NURS 6501 Advanced pathophysiology: In the week's case study, a 27 year old was presented with a history of substance abuse and was found unresponsive by EMS after being called by the patient's roommate. Unknown as to how long the patient had been lying on there, the patient was administered naloxone and became responsive. The patient complained of burning pain over the left hip and forearm. Further work up in the ED revealed a large amount of necrotic tissue over the patients greater trochanter and forearm and an EKG demonstrating a prolonged PR interval, peaked T waves and a serum potassium level of 6.9 mEq/L.
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Intracellular Potassium Shifts
Cellular injury can release large quantities of intracellular potassium into the extracellular space. This can be due to rhabdomyolysis from a crush injury, excessive exercise, or other hemolytic processes.
Explanation: Increased tissue breakdown such as rhabdomyolysis, burns, or any cause of rapid tissue necrosis, including tumor lysis syndrome can cause the release of intracellular potassium into blood, causing hyperkalemia.
Patients with chronic hyperkalemia may have relatively normal EGCs even at high levels, and significant ECG changes may be present at much lower levels in patients with sudden spikes in serum potassium.ECG features of hyperkalemia include:
- Small or absent P wave
- Prolonged PR interval
- Augmented R wave
- Wide QRS
- Peaked T waves
